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These critical capabilities could also be associated to MOF-mediated acetylation of chromatin-associated proteins other than H4 or alterations of chromatin construction induced by such modifications that are not detectable by DNaseI sensitivity.
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As an illustration, human SET7/9 has just lately been reported to methylate Lys189 within the normal transcription factor TAF10, resulting in an increased affinity for RNA polymerase II and transcriptional activation of sure TAF10-dependent genes 37 SET7/9 has additionally been reported to [methylate](https://slicethis.link/sybilt66170364) p53, rising the soundness of this brief lived tumor-suppressor protein 38 These observations suggest that we should not narrowly outline the SET-domain proteins as histone lysine methyltransferases however instead call them protein lysine methyltransferases.
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